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cascade, sustaining the anti-inflammatory/anti-fibrotic phenotype by the secretion of anti-inflammatory cytokines, such as IL-10. Furthermore, Tregs are in a position to improve healing quality by inducing a sort 2 response, together with anti-inflammatory macrophages. Following a T cells decrease, resident Treg ranges stay elevated, probably as a result of they show an epithelial development factor receptor (EGF-R) whose expression allows the expansion factor amphiregulin secreted by mast cells to take care of Tregs on the damaged site . Once present, Tregs proliferate and upregulate the secretion of amphiregulin, which may induce both cell

 

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